THEY KNEW ABOUT CHLOROQUINE IN 2004! AND 2005! AND 2006!
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Remember in the year 2020 when the top infectious disease expert, Dr. Fauci, opposed hydroxychloroquine as an unproven treatment?
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And instead pushed for waiting it out until remdesivir and a vaccine are approved, both of which his institution partnered on?
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Ironically, the same day in February (the 25th), that French doctor Didier Raoult said it was “Game Over” for coronavirus because he has done studies with great success using hydroxychloroquine, that was the same day that Fauci instead launched a clinical trial for remdesivir.
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Just like the same day a New York doctor, Dr. Zev Zelenko, published a “Success” report using hydroxychloroquine, Governor Cuomo that same day restricted the use of hydroxychloroquine (March 24).
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Anyways, back to my original point. The year 2020 when Fauci said there was no evidence that hydroxychloroquine or chloroquine was effective in treating SARS-CoV-2…. well as it turns out, there were studies published on the NIH own website, dating back to October, 8th, 2004; August 22, 2005; and November 28th, 2006, all stating that chloroquine was effective because it inhibits the ACE2 receptor.
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We knew as early as February 3, 2020 that SARS-CoV-2 uses hACE2 as the receptor because a Chinese scientist named Peng Zhou reported that.
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Yet, while Fauci started clinical trial for remdesivir on February 25th, it wasn’t until April 9th that he started a clinical trial for hydroxychloroquine.
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Read the 2004, 2005, and 2006 research yourself:
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October 8th, 2004: An article is published on a NIH website entitled, In vitro inhibition of severe acute respiratory syndrome coronavirus by chloroquine.”
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They said, “We report on chloroquine, a 4-amino-quinoline, as an effective inhibitor of the replication of the severe acute respiratory syndrome coronavirus (SARS-CoV) in vitro. Chloroquine is a clinically approved drug effective against malaria. We tested chloroquine phosphate for its antiviral potential against SARS-CoV-induced cytopathicity in Vero E6 cell culture. Results indicate that the IC50 of chloroquine for antiviral activity (8.8 +/- 1.2 microM) was significantly lower than its cytostatic activity; CC50 (261.3 +/- 14.5 microM), yielding a selectivity index of 30. The IC50 of chloroquine for inhibition of SARS-CoV in vitro approximates the plasma concentrations of chloroquine reached during treatment of acute malaria. Addition of chloroquine to infected cultures could be delayed for up to 5h postinfection, without an important drop in antiviral activity. Chloroquine, an old antimalarial drug, may be considered for immediate use in the prevention and treatment of SARS-CoV infections.” https://www.ncbi.nlm.nih.gov/pubmed/15351731
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August 22, 2005: American Scientists publish a paper on the NIH website, entitle, “Chloroquine is a potent inhibitor of SARS coronavirus infection and spread.”
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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1232869/
“Severe acute respiratory syndrome (SARS) is caused by a newly discovered coronavirus (SARS-CoV). No effective prophylactic or post-exposure therapy is currently available… We report, however, that chloroquine has strong antiviral effects on SARS-CoV infection of primate cells. These inhibitory effects are observed when the cells are treated with the drug either before or after exposure to the virus, suggesting both prophylactic and therapeutic advantage. In addition to the well-known functions of chloroquine such as elevations of endosomal pH, the drug appears to interfere with terminal glycosylation of the cellular receptor, angiotensin-converting enzyme 2. This may negatively influence the virus-receptor binding and abrogate the infection, with further ramifications by the elevation of vesicular pH, resulting in the inhibition of infection and spread of SARS CoV at clinically admissible concentrations… Chloroquine is effective in preventing the spread of SARS CoV in cell culture. Favorable inhibition of virus spread was observed when the cells were either treated with chloroquine prior to or after SARS CoV infection. In addition, the indirect immunofluorescence assay described herein represents a simple and rapid method for screening SARS-CoV antiviral compounds.” https://virologyj.biomedcentral.com/articles/10.1186/1743-422X-2-69
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November 28th, 2006: An article is published on a NIH website, entitled, “Severe Acute Respiratory Syndrome Coronavirus (SARS-CoV)”
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“Chloroquine, discovered by the German chemist Hans Andersag in 1934 and used for the treatment of malaria, amebiasis, HIV, and autoimmune diseases, was recognized to have activity against SARS-CoV in vitro, with a selectivity index of 30 against SARS-CoV in Vero E6 cell culture (Savarino et al., 2003; Keyaerts et al., 2004; Vincent et al., 2005). Chloroquine elevates endosomal pH and interferes with terminal glycosylation of ACE2 (Vincent et al., 2005), thus having both non-specific and specific anti-SARS-CoV activities.”
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In other words, Chloroquine works by helping to block the ACE2 receptor from connecting with the coronavirus, which the virus needs for cellular entry for infection.
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A little Drug History:
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The NIAID, Fauci’s Institution, is the trial sponsor for Gilead, who developed remdesivir.
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Fauci’s Institution partnered with them. Which explains why he opposed hydroxychloroquine. It’s the competitor.
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Chloroquine was shown to be effective against SARS-CoV back in 2004, 2005, 2006.
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But Fauci launched clinical trials for remdesivir in February. And hesitantly dragged his feet to launch a clinical trial for hydroxychloroquine in April. Months after doctors around the world reported its effectiveness.
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And strangely, even though reports are saying remdesivir had no significant benefit to patients, and doctors are demanding more evidence, Fauci is touting this new drug as a game changer.
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Ironically, the same grant money used to fund the Wuhan Lab, Fauci used to fund the clinical trial for Remdesivir. That’s the ecohealth grant.
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And the Wuhan Lab, January 31, filed a patent for the use of remdesivir and chloroquine as a treatment together.
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Even the Wuhan Lab knew about chloroquine.
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January 31, 2020: The Wuhan Institute of Virology files for a patent regarding Remdesivir, a drug treatment for Covid-19.
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“Soon after receiving Remesdivir samples from Gilead, the Wuhan Institute of Virology in China (“Wuhan Institute”) filed a competing “method of use” patent that involves using Remdesivir in conjunction with chloroquine, a common malaria antiviral.”
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February 4, 2020: Shi Zhengli posted on the Wuhan Institute of Virology, “our findings reveal that remdesivir and chloroquine are highly effective in the control of 2019-nCoV infection in vitro. Since these compounds have been used in human patients with a safety track record and shown to be effective against various ailments, we suggest that they should be assessed in human patients suffering from the novel coronavirus disease.”
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Chloroquine “inhibited SARS-CoV-2 infection in vitro” meaning it blocked the S Spike Protein of the virus from connecting with the human ACE2 enzyme, by “acting as a weak base and immunomodulator”
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January 31st. February 4th. That was before any major announcements in America were made.
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Since Dr. Fauci funded the Wuhan Lab, he sponsored Remdesivir, He partnered with the Gates Vaccine, and he sits on the Leadership Council for vaccines for the Gates Foundation, it is clear that Dr. Fauci has a “conflict of interest” in all things relating to this pandemic from Wuhan.
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Even the Wuhan Lab knew.
January 31, 2020: The Wuhan Institute of Virology files for a patent regarding Remdesivir, a drug treatment for Covid-19.
“Soon after receiving Remesdivir samples from Gilead, the Wuhan Institute of Virology in China (“Wuhan Institute”) filed a competing “method of use” patent that involves using Remdesivir in conjunction with chloroquine, a common malaria antiviral.”
February 4, 2020: Shi Zhengli posted on the Wuhan Institute of Virology, “our findings reveal that remdesivir and chloroquine are highly effective in the control of 2019-nCoV infection in vitro. Since these compounds have been used in human patients with a safety track record and shown to be effective against various ailments, we suggest that they should be assessed in human patients suffering from the novel coronavirus disease.”
Chloroquine “inhibited SARS-CoV-2 infection in vitro” meaning it blocked the S Spike Protein of the virus from connecting with the human ACE2 enzyme, by “acting as a weak base and immunomodulator”
January 31st. February 4th. That was before any major announcements in America were made.
Isn’t it strange that on January 26th, 2020, when Dr. Fauci said that coronavirus was a “very, very low risk to the United States”, he also said that he was already working on a vaccine for it?
Why would you invest millions of dollars into a vaccine that you say was very very low risk? The global death toll at that point was only 80 people.
It’s just strange that he downplayed the risk of the coronavirus at the beginning, even being opposed to Trump’s travel ban, and thus Fauci helped enabled the coronavirus to travel here and spread. And then as soon as it arrived and started spreading, he said we need to shut down the economy until there is a vaccine.
It’s just very suspicious to me.
Isn’t it strange that on January 26th, 2020, when Dr. Fauci said that coronavirus was a “very, very low risk to the United States”, he also said that he was already working on a vaccine for it?
Why would you invest millions of dollars into a vaccine that you say was very very low risk? The global death toll at that point was only 80 people. In fact, the day he started the vaccine, January 20th, there were zero deaths.
It’s just strange that he downplayed the risk of the coronavirus at the beginning, even being opposed to Trump’s travel ban, and thus Fauci helped enabled the coronavirus to travel here and spread. And then as soon as it arrived and started spreading, he said we need to shut down the economy until there is a vaccine, which he already had in the works?
It’s just very suspicious to me.
It was his job to know. Of course he knew.